Extracellular matrix components induce endocrine differentiation in vitro in NCI-H716 cells. - Abstract - Europe PMC
![A, Immunocytochemical analysis of NCI-H716 cells using antisera against... | Download Scientific Diagram A, Immunocytochemical analysis of NCI-H716 cells using antisera against... | Download Scientific Diagram](https://www.researchgate.net/publication/7496334/figure/fig1/AS:394492671741960@1471065790938/A-Immunocytochemical-analysis-of-NCI-H716-cells-using-antisera-against-GLP-1-GLU.png)
A, Immunocytochemical analysis of NCI-H716 cells using antisera against... | Download Scientific Diagram
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
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Amazon.com : Mild Neon Color Daylight Fluorescent ink Highlighter for students (H716) : Office Products
![Peptide production and secretion in GLUTag, NCI-H716, and STC-1 cells: a comparison to native L-cells in: Journal of Molecular Endocrinology Volume 56 Issue 3 (2016) Peptide production and secretion in GLUTag, NCI-H716, and STC-1 cells: a comparison to native L-cells in: Journal of Molecular Endocrinology Volume 56 Issue 3 (2016)](https://jme.bioscientifica.com/view/journals/jme/56/3/images/large/201fig3.jpeg)
Peptide production and secretion in GLUTag, NCI-H716, and STC-1 cells: a comparison to native L-cells in: Journal of Molecular Endocrinology Volume 56 Issue 3 (2016)
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
![The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S1347861320301110-gr5.jpg)
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
![The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S1347861320301110-gr2.jpg)
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
![The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S1347861320301110-gr4.jpg)